Studies have clearly shown that your immune cells weaken as you age. Scientists have discovered that telomere shortening plays an important role in this aging process.

The February 2013 issue of the Journal of the American Medical Association (JAMA)reports an association between decreased telomere length and greater susceptibility to the common cold. The work suggests that the length of telomeres can predict risks for people as young as 22; and the association only gets stronger with age.

To gauge exactly how those protective caps work, lead researcher Sheldon Cohen of Carnegie Mellon University and his team measured the telomere length of white blood cells from 152 healthy volunteers aged 18-55. They were later exposed to the rhinovirus, responsible for the common cold, and quarantined in single rooms for five days to see if infections would develop.

Among 105 infected with the virus, 33 people developed colds. And individuals with shorter telomeres were more likely to get respiratory infections, although this was only true among those aged 21 or older. Overall, people with shorter telomeres in a specific type of blood cell – a CD8CD28 T-cell that is critical for eliminating infections — were more likely to succumb to infections.

The immune system falls apart with age in ways that are as much a matter of configuration as wear and tear – it is a machine in which the programming runs awry, leading it to do the wrong things at the wrong time, or just do nothing when it should be doing something. This activity leads to damage, which in turn accelerates aging: “Immune aging is associated with loss of critical immune functions, such as host protection from infection and malignancy. Unexpectedly, immunosenescence also renders the host susceptible to inflammation, which may translate into tissue-damaging disease as the senescent immune system loses its ability to maximize inflammatory protection while minimizing inflammatory injury.

Inflammation is a major culprit behind most serious health issues including your joints, blood vessels, immunity, metabolic health. But now we are now gaining important insights into the causes of inflammation, and perhaps how you can fight back.

A new, compelling inflammation study from the University of California at San Francisco gives us clear evidence that telomere length is the key. It determines your risk and the severity of your inflammation.

In the study, researchers looked at 1,962 healthy men and women between the ages of 70 and 79. Those with short telomeres had high levels of two pro-inflammatory factors interleukin-6 (IL-6)and tumor necrosis factor alpha (TNF-a).

Both IL-6 and TNF-a are known as cytokines, a group of compounds that cause chronic inflammation.

But cytokines play another harmful role in your body – they block telomerase, the enzyme that builds your telomeres.

This is doubly dangerous for your body. First, your tissues become inflamed. Then, your telomeres get shorter, making your cells older and weaker.

Luckily, we now have evidenced-based nutrients and antioxidants that repair and lengthen your telomeres so you can fight back against these destructive biological processes. So now, by maintaining the length of your telomeres, you can address inflammation and the chronic health issues that go along with it.

You can now create a younger, happier, more energetic version of yourself.

Please explore our website TeloVite.com and learn about the 14 telomere lengthening nutrients and antioxidants in the broad-spectrum, high potency TeloVite multivitamin, the first Cellular Longevity Multivitamin, with ingredients backed by 312 peer-reviewed scientific studies.